Mycoplasma penetrans infection of Molt-3 lymphocytes induces changes in the lipid composition of host cells
Article Abstract:
A study was undertaken to investigate the phospholipid degradation in Molt-3 lymphocytes infected by Mycoplasma penetrans and to identify the lipid intermediates produced. Mycoplasmas bind themselves to the host-cell surface preceded by internalization. Results indicated that infection of Molt-3 lymphocytes by M. penetrans led to the substantial production of fatty acids which indicated alterations in the lipid turnover of the host cells. The infection was related to the release of the lipid intermediates acting as signal transductors across the host-cell membrane.
Publication Name: Microbiology
Subject: Biological sciences
ISSN: 1350-0872
Year: 1998
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Molecular and biochemical analysis of a 105 kDa Mycoplasma gallisepticum cytadhesin (gapA)
Article Abstract:
Research was conducted to examine the molecular and biochemical properties of a 105 kDa Mycoplasma gallisepticum cytadhesin (GapA). The objectives of the study were to characterize the gapA gene and its encoded protein and to analyze the functional role of GapA in cytadherence. DNA sequencing, restriction enzyme analysis and Southern blot hybridization were used in the identification of the gapA gene. Results demonstrated that the gapA gene is present in many different strains of M gallisepticum.
Publication Name: Microbiology
Subject: Biological sciences
ISSN: 1350-0872
Year: 1998
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Lymphocytic infiltration in the chicken trachea in response to Mycoplasma gallisepticum infection
Article Abstract:
Lymphocytic infiltration as a response to Mycoplasma gallisepticum infection in chicken trachea is discussed. Numbers and distribution of lymphoctyes in the tracheas of chickens infected with a virulent strain of M. gallisepticum have been studied. The study is the first to look at phenotypes of T lymphocytes infiltrating respiratory tracts in mycoplasma infections. It appears that involvement of specific stimulation of CD8+ cells exists, especially in the acute disease phase.
Publication Name: Microbiology
Subject: Biological sciences
ISSN: 1350-0872
Year: 2000
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