A seedling lethal maize mutant lacking functional leaf stomates
Article Abstract:
The lack of functional stomates due to the expression of the recessive abnormal stomates 1 gene (abs1) causes the lethality in seedling lethal maize mutant. The segregation patterns of abs1 in the F(sub 2) and F(sub 3) generations indicate that the phenotype is caused by a single recessive gene. The stomates in the mutant plants lack two subsidiary cells and normal guard cells. This indicates that abs1 gene inhibits normal stomatal development. The dry weight, nonstructural carbohydrates and chlorophyll content of the mutants is less than that of normal seedlings. The water and nitrate content of the mutants is higher due to the lack of transpiration.
Publication Name: The Journal of Heredity
Subject: Biological sciences
ISSN: 0022-1503
Year: 1996
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Microsatellite repeat variation within the y1 gene of maize and teosinte
Article Abstract:
The (CCA)(sub n) microsatellite containing region of the y1 gene varies in repeat number from three to 11 in six teosinte species, subspecies or varieties; and 11 different maize lines. An imperfect pentanucleotide repeat is also present adjacent to this trinucleotide microsatellite region. The variation in the number of repeats and the flanking region in maize and teosinte can provide markers in molecular biology studies. The y1 gene regulates the carotenoids content of the maize kernels and the markers can be used to monitor the carotenoid content.
Publication Name: The Journal of Heredity
Subject: Biological sciences
ISSN: 0022-1503
Year: 1996
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Biochemical and ultrastructural analysis of the y10 mutant of maize
Article Abstract:
The pale-yellow endosperm colour of the y10 mutant of maize and its albino seedling categorize it as a carotenoid mutant. However, y10 seedlings have not been seen to become pale green when grown in reduced light, unlike other carotenoid-deficient mutants. A new study seeks to characterize the biochemical defect present in the y10 mutant phenotype and to determine its effect on the structure of plastids in such mutants. Details of the study are presented.
Publication Name: The Journal of Heredity
Subject: Biological sciences
ISSN: 0022-1503
Year: 1999
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