Common biological pathways in eating disorders and obesity

Article Abstract:

Dysfunctions in the serotonin and beta-endorphin systems show the greatest potential for trait biological etiologies in aneroxia nervosa, bulimia nervosa (BN) and obesity. These systems can play crucial roles in all three disorders, with biologically plausible mechanisms. Persons experiencing BN may have lower levels of serotonin and may eat foods high in tryptophan, a serotonin precursor, to make up for the deficiency. On the other hand, beta-endorphin levels may be high in BN patients and may lead to appetite stimulation, bingeing and vomiting. Once stabilized, their beta-endorphin levels may decline and help trigger a relapse.

Physiological aspects, Obesity, Anorexia nervosa

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Parental variables associated with bulimia nervosa

Article Abstract:

A group of 2,315 undergraduate psychology females and their parents were examined to test daughter/parent variables such as attitudes about body image, eating and dieting histories and self esteem, associated with bulimia nervosa. The sample was divided into three groups; those who met all five DSM-III-R criteria were bulimic group, women who met 1-4 criteria constituted the sub clinical group and women who met no criteria constituted the control group. Bulimic daughters felt that mothers had been pressurizing them to lose weight.

Research, Mothers and daughters, Mother-daughter relations

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Psychological factors in the etiology of binge eating

Article Abstract:

It has been hypothesized that several psychological factors are associated with the etiology of binge eating. There is a proposal that female gender-role socialization is a risk factor for the development of binge eating in girls. To understand this risk, a study of the developmental tasks of female adolescence is necessary. Research of the etiology of binge eating should focus on complex and multifactorial models of causation instead of single-factor causal models.

Psychological aspects, Diseases, Etiology (Medicine)

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