One or two hours postprandial glucose measurements: are they the same?
Article Abstract:
Women with diabetes during pregnancy will reach maximal blood sugar values one hour after breakfast but two hours after dinner. This could occur because dinner contains more protein and fat than breakfast. Diabetics may want to wait until two hours after dinner to measure their blood sugar levels.
Publication Name: American Journal of Obstetrics and Gynecology
Subject: Health
ISSN: 0002-9378
Year: 2001
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Fetal Doppler and behavioral responses during hypoglycemia induced with the insulin clamp technique in pregnant diabetic women
Article Abstract:
Brief episodes of moderate low blood sugar do not appear to affect the fetus of insulin-dependent diabetic women. While it is known that tight metabolic control improves pregnancy outcomes among insulin-dependent diabetics, this results in frequent episodes of hypoglycemia. Ten insulin-dependent diabetic women at gestational ages ranging from 27 to 37 weeks underwent a controlled reduction of blood sugar by infusing insulin. Maternal blood sugar was reduced to 45 mg/dl for 40 minutes. Electronic fetal monitoring and ultrasound imaging evaluated fetal heart rate, fetal practice breathing movements (a measure of fetal well-being), and fetal limb movements. Placental perfusion was evaluated via Doppler ultrasound studies as well. No adverse fetal effects were found. The effects of long-term or severe hypoglycemia on the fetus has not been studied.
Publication Name: American Journal of Obstetrics and Gynecology
Subject: Health
ISSN: 0002-9378
Year: 1995
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Dietary vitamin E prophylaxis and diabetic embryopathy: morphologic and biochemical analysis
Article Abstract:
Vitamin E supplements may protect against fetal maldevelopment in diabetic rats. High blood sugar causes congenital defects of the brain, spinal cord, and heart apparently by increasing oxidative compounds. Vitamin E is an antioxidant. Of three groups of diabetic rats, one received dietary vitamin E supplements, one ate a normal diet, and one received insulin therapy. Supplemented rats had spinal cord and brain defect rates and embryonic resorption rates similar to that of normal rats and rats whose blood sugar was controlled with insulin. Untreated, unsupplemented rats had markedly increased incidences of defects and embryonic resorption.
Publication Name: American Journal of Obstetrics and Gynecology
Subject: Health
ISSN: 0002-9378
Year: 1996
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